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Trenbolone acetate vs Trenbolone Enanthate would be the same thing as comparing testosterone prop (a short ester) to testosterone enanthate (a longer acting ester)(which isn't something we do). We don't have to compare Tren to Tren to make this distinction. They're not both short-acting. We don't know that the active testosterone is longer-acting, because testosterone is a molecule with multiple chemical and biological effects, many of which can be inhibited by longer-acting steroid. I'm sure some of the people who think testosterone is a longer-acting testosterone would just make a bunch of silly, absurd comparisons, that aren't what these trials show. I have been a proponent of Tren and Trenbolone Enanthate as therapy for many years, and I think that Tren in general is an amazing compound. I believe it could make a dramatic improvement in many chronic degenerative diseases, and I'm thrilled that our team has put us on such excellent footing with all of our patients around the country. There will be more in the future - that's when we'll really start to get a handle on it. Right now, the best information is based on animal studies in dogs and rodents, as well as clinical trials with people. In terms of people's experiences with this medication, I'm not aware of any other drug that has been demonstrated to dramatically reduce the symptoms of multiple sclerosis. Why are Trenbolo/Enanthate treated with a different formulation of progesterone? Would it be better to take Enanthate from the day of therapy, or would it work better the next day? This would be a very well-known protocol, but you probably just don't want to learn about it from my mouth, so I'll leave it to your expert knowledge of progesterone: In order to help the body absorb the steroid, there are multiple options for how to achieve this. First is to change the way we create progesterone for use in the body, as opposed to a synthetic form which is used internally. It's pretty easy to work around in the body: we have a process for adding progesterone to animal milk that can be easily altered for different patients. This is what happens when a synthetic hormone is added to a cow's milk: its insulin can be increased in order to ensure proper growth. That would make sense in humans, but there are two issues to be aware of: first, that is not a reliable process for adding progesterone to human milk, and second, the human body doesn't naturally make it. So, we need a similar method to make human prog Similar articles:

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